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Dramatic developments in genetics research and the availability of commercial genetics tests have put us in a very modern predicament—we can now find out (quickly, easily and cheaply) whether we personally hold genetic risk factors that put us at a substantially increased risk of Alzheimer's disease.
A team of scientists from the Moscow Institute of Physics and Technology (MIPT) and Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry (IBCh RAS) studied one hereditary genetic mutation to discover general molecular mechanisms that may lead both to early onset of Alzheimer's disease and to the form of the disease caused by age-related changes in human body.
A gene that can become mutated and cause a rare balance disorder also regulates the behavior of an enzyme that increases the risk for Alzheimer's disease (AD), according to a new study by researchers at Massachusetts General Hospital (MGH) to be published in the journal Cell on August 22, 2019.
Protein aggregation—in which misfolded proteins clump together to form large fibrils—has been implicated in many diseases including Alzheimer's, Parkinson's, and type II diabetes. While the exact role these fibrils play in diseases isn't fully understood, many of the current treatments for diseases like Alzheimer's and Parkinson's target the aggregation process. However, finding the right treatment protocols for these drugs, which can be toxic in large doses, is challenging.
The idea of seeing a loved one decline and lose their ability to recall their most treasured memories is devastating. However, it is a fact of life for an increasing number of Canadians. A group of experts on population health convened by the Alzheimer Society of Canada in 2015 estimated that nearly one million Canadians will have Alzheimer's disease in 2031.
Alzheimer's Research UK is calling on the Prime Minister to back worldwide commitments to invest in more dementia research as new data out today shows deaths due to the condition continue to rise. According to the Office of National Statistics, Alzheimer's disease and other dementias remain the leading cause of death in England and Wales and accounted for 12.8 percent of all deaths in 2018.
Memory impairment and mood changes are typically observed in patients with Alzheimer's disease but what disturbs these neuronal functions is unclear. Some researchers have proposed that alterations in the production of new neurons in the brain, or neurogenesis, may be involved; however, whether neurogenesis happens in humans, much less those with Alzheimer's disease, has been debated. A discovery published today in the journal Cell Reports provides a possible explanation for this debate and may shed light on what happens in Alzheimer's disease.

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