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Oral Diseases 10/20/2019 12:18
Abstract. Background. Buccal mucosal squamous cell carcinoma (BMSCC) is an aggressive oral cancer. Moreover, reversion‐inducing cysteine‐rich protein with Kazal motifs (RECK) is a well‐known tumor suppressor in many cancers. Our aim was to investigate the association of RECK expression with prognosis in BMSCC patients with different clinicopathological features. Materials and Methods. The expression level of RECK was determined by immunohistochemistry using tissue microarrays containing specimens from 193 BMSCC patients. The association of RECK expression with outcomes in BMSCC patients stratified by different clinicopathological features was analyzed by Cox proportional hazards models. Results. The low expression level of RECK was associated w.
Oral Diseases 10/19/2019 02:19
Abstract. Objective. This study was conducted to distinguish salivary metabolites in oral squamous cell carcinoma (OSCC) from those in oral lichen planus (OLP) to identify practical biomarkers for the discrimination of OSCC from OLP. Subjects and methods:. Whole unstimulated saliva samples were collected from patients with OSCC (n = 34) and OLP (n = 26).
Business Wire 10/18/2019 07:00
Head and Neck Cancer KEYTRUDA, in combination with platinum and fluorouracil (FU), is indicated for the first-line treatment of patients with metastatic or with unresectable, recurrent head and neck squamous cell carcinoma (HNSCC.
Oral Diseases 10/16/2019 11:19
Abstract. Objective. Recent studies have demonstrated the pro‐tumour role of CD36 in multiple cancer types. However, its role has not been well elucidated in oral squamous cell carcinoma (OSCC). Here, we aimed to evaluate the role of CD36 in proliferation and migration of OSCC cells. Methods. Human OSCC cell lines HSC‐2, HSC‐3, HSC‐4, and Ca9‐22 were assessed for proliferation by staining with the cell proliferation marker Ki‐67. We also assessed migration activity, and the expression of cell adhesion molecules such as E‐cadherin and β‐catenin and platelet‐derived growth factor receptors (PDGFRs) of CD36‐positive cells. Results. CD36‐positive cells showed increased expression of Ki‐67 and migration activity compared with CD36‐negative cells. Mo.

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