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FeedNavigator (EU) 09/16/2019 13:37
A study has found a protein which, when depleted, resulted in Alzheimer’s disease symptoms appearing in mouse and brain tissue models.
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Brain tissue of Alzheimer’s disease patients invariably contains deposits of insoluble, fibrillar aggregates of peptide fragments of the amyloid precursor protein (APP), typically 40 or 42 residues in length and referred to as Aβ40 and Aβ42.
More from Journal of the American Chemical Society: Latest Articles (ACS Publications):
Fight Aging! 08/20/2019 06:22
Alzheimer's patients often experience damage to brain blood vessels caused by amyloidproteins, an effect known as cerebral amyloid angiopathy (CAA.
More from Fight Aging!: 08/13/2019 10:20
In search of a possible cure for Alzheimer’s disease (AD), scientists have found out the molecular structure of a type of protein called beta amyloid that accumulates within brain cells, eventually killing them.
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Technology Org 08/08/2019 05:30
The researchers concluded that fluorodeoxyglucose (FDG) PET, which measures the brain’s glucose consumption as a marker of neural activity, is a stronger approach for assessing the progression and severity of Alzheimer’s and mild cognitive impairment (MCI) as compared to florbetapir-PET scans, which reveal amyloid protein deposits in the brain.
More from Technology Org:
The most common form of dementia, Alzheimer’s disease (AD), is characterized by the accumulation of senile β-amyloid (Aβ) peptide plaques and intracellular neurofibrillary tangles.
More from Science Translational Medicine:
EurekAlert 07/17/2019
This multicenter study used in vivo β-amyloid cerebrospinal fluid, a biomarker of Alzheimer disease, and positron emission tomography findings to track progression of Alzheimer disease over six years among study participants.
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