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GlobeNewswire 09/13/2019 15:58
SHANGHAI, Sept. 13, 2019 (GLOBE NEWSWIRE) -- Xynomic Pharmaceuticals Holdings, Inc. ("Xynomic", stock ticker: XYNO), a clinical stage US-China oncology drug development company, today announced the dosing of the first Chinese patient in its on-going global pivotal Phase 3 trial of Xynomic’s abexinostat combined with pazopanib as a first- or second-line therapy against renal cell carcinoma (RCC) at Peking University Cancer Hospital & Institute.
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Silencing circZFR inhibits the proliferation, migration and invasion of human renal carcinoma cells by regulating miR-206.
More from International Journal of COPD: 09/12/2019 07:48
Condition : Renal Cell Carcinoma Interventions : Biological: Nivolumab; Biological: Ipilimumab Sponsor : Bristol-Myers Squibb Not yet recruiting.
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Business Wire 09/10/2019 06:30
Tivozanib has been investigated in several tumor types, including renal cell, hepatocellular, colorectal, ovarian and breast cancers.
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Healio News 09/09/2019 05:30
Austin G. Stack Gout is associated with a 29% increased risk for developing advanced chronic kidney disease and a 210% increased risk for end-stage renal disease, according to data published in BMJ Open.
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FeedNavigator (EU) 09/07/2019 08:52
This review also details progress made in understanding the effect of sirtuins in the pathophysiology of chronic and acute kidney diseases, highlighting the key role of SIRT1, SIRT3, and now SIRT6 as potential therapeutic targets.
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Cancer Cell International 09/06/2019 20:00
MicroRNA-200c-3p (miR-200c-3p) has been revealed to be related to renal cell carcinoma (RCC) progression, while the inner mechanisms remain unknown. In our study, we intend to unearth the capability of miR-200...
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Clear cell renal cell carcinoma (ccRCC) is the most common subtype of kidney cancer. While the localized form of this disease can be treated surgically, advanced and metastatic stages are resistant to chemotherapies. Although more innovative treatments, such as targeted or immune-based therapies, exist, the need for new therapeutic options remains. ccRCC presents unique metabolic signatures and multiple studies have reported a significant increase in levels of reduced glutathione (GSH) and its precursors in ccRCC tumor samples compared with normal kidney tissues. These observations led us to investigate the effects of blocking the GSH pathway, particularly the gamma-glutamyltransferase 1 (GGT1) enzyme, in multiple ccRCC cell lines. In this

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